Import filter:
ris_refman.bif (32.8 KB)
Last update:
20060616
Provider:
Thomson ResearchSoft
Comments:
This is an attemp to create an import filter for RIS that works well for all RIS files from different sources and versions.
Example:
TY - JOUR
A1 - Baldwin,S.A.
A1 - Fugaccia,I.
A1 - Brown,D.R.
A1 - Brown,L.V.
A1 - Scheff,S.W.
T1 - Blood-brain barrier breach following cortical contusion in the rat
JO - J.Neurosurg.
Y1 - 1996
VL - 85
SP - 476
EP - 481
RP - Not In File
KW - cortical contusion
KW - blood-brain barrier
KW - horseradish peroxidase
KW - head trauma
KW - hippocampus
KW - rat
N2 - Adult Fisher 344 rats were subjected to a unilateral impact to the
dorsal cortex above the hippocampus at 3.5 m/sec with a 2 mm cortical
depression. This caused severe cortical damage and neuronal loss in
hippocampus subfields CA1, CA3 and hilus. Breakdown of the blood-brain
barrier (BBB) was assessed by injecting the protein horseradish peroxidase
(HRP) 5 minutes prior to or at various times following injury (5 minutes, 1,
2, 6, 12 hours, 1, 2, 5, and 10 days).Animals were killed 1 hour after HRP
injection and brain sections were reacted with diaminobenzidine to visualize
extravascular accumulation of the protein. Maximum staining occurred in
animals minutes after cortical contusion. Staining at these time points was
observed in the ipsilateral hippocampus. Some modest staining occurred in the
dorsal contralateral cortex near the superior sagittal sinus. Cortical HRP
stain gradually decreased at increasing time intervals postinjury. By 10
days, no HRP stain was observed in any area of the
brain. In the ipsilateral hippocampus, HRP stain was absent by 3 hours
postinjury and remained so at the 6- and 12- hour time points. Surprisingly,
HRP stain was again observed in the ipsilateral hippocampus 1 and 2 days
following cortical contusion, indicating a biphasic opening of the BBB
following head trauma and a possible second wave of secondary brain damage
days after the contusion injury. These data indicate regions not initially
destroyed by cortical impact, but evidencing BBB breach, may be accessible to
neurotrophic Reference Manager User’s Guide 369
factors administered intravenously both immediately and days after brain
trauma.
ER -
TY - PAT
A1 - Burger,D.R.
A1 - Goldstein,A.S.
T1 - Method of detecting AIDS virus infection
Y1 - 1990/2/27
VL - 877609
IS - 4,904,581
RP - Not In File
A2 - Epitope,I.
CY - OR
PB - 4,629,783
KW - AIDS
KW - virus
KW - infection
KW - antigens
Y2 - 1986/6/23
M1 - G01N 33/569 G01N 33/577
M2 - 435/5 424/3 424/7.1 435/7 435/29 435/32 435/70.21 435/240.27 435/172.2
530/387 530/808 530/809 935/110
N2 - A method is disclosed for detecting the presence of HTLV III infected
cells in a medium. The method comprises contacting the medium with monoclonal
antibodies against an antigen produced as a result of the infection and
detecting the binding of the antibodies to the antigen. The antigen may be a
gene product of the HTLV III virus or may be bound to such gene product. On
the other hand the antigen may not be a viral gene product but may be
produced as a result of the infection and may further be bound to a
lymphocyte. The medium may be a human body fluid or a
culture medium. A particular embodiment of the present method involves a
method for determining the presence of a AIDS virus in a person. The method
comprises combining a sample of a body fluid from the person with a
monoclonal antibody that binds to an antigen produced as a result of the
infection and detecting the binding of the monoclonal antibody to the
antigen. The presence of the binding indicates the presence of a AIDS virus
infection. Also 370 Appendix C—RIS Format Specifications disclosed are novel
monoclonal antibodies, noval compositions of matter, and novel
diagnostic kits
ER -